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Glycation, Aging, and Low Carbohydrate Diets
Koji Ebe1,2, Michael Wood1, Hiroshi Bando1,3iD*
1Japan Low Carbohydrate Diet Promotion Association (JLCDPA), Kyoto, Japan
2Takao Hospital, Kyoto, Japan
3Medical Research/Tokushima University, Tokushima, Japan
Corresponding Author: Hiroshi Bando ORCID iD
Address: Tokushima University /Medical Research, Nakashowa 1-61, Tokushima 770-0943, Japan.
Received date: 26 March 2025; Accepted date: 16 April 2025; Published date: 23 April 2025
Citation: Ebe K, Wood M, Bando H. Glycation, Aging, and Low Carbohydrate Diets. Asp Biomed Clin Case Rep. 2025 Apr 23;8(1):85-88.
Copyright © 2025 Ebe K, Wood M, Bando H. This is an open-access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium provided the original work is properly cited.
Keywords: Low-Carbohydrate Diet, Advanced Glycation End Products, Glycation, Longevity
Abbreviations: LCD: Low-Carbohydrate Diet; AGEs: Advanced Glycation End Products
Abstract
The term "AGEs" is an acronym derived from "Advanced Glycation End Products (AGEs)." The medical community has been focusing on AGEs in recent years. So, what exactly is "glycation"? Glycation refers to the reaction in which monosaccharides such as glucose or fructose directly bind to proteins or other substances. AGEs produced in the body cause diabetic complications. AGEs have garnered attention because they are now considered a primary cause of various diabetic complications. The processes involved are diverse, but the most straightforward example is when AGEs accumulate in the inner walls of blood vessels, leading to atherosclerosis. Depending on which part of the blood vessels is affected by atherosclerosis, the resulting diabetic complications vary, but it is no exaggeration to call them all vascular diseases. AGEs in the vessel walls are like an unpayable debt, often referred to as the "memory of high blood sugar."